Disclaimer; I am not a medical professional. These are my findings and conclusions based on anecdotal and medical evidence.
 I have conducted no medical research of my own.
Some parts of this paper containing are copied and pasted from medical journals, as there is no need for me to rewrite them.
I have rewritten all else as I understand it just to keep it digestible and layman-friendly.


08/Aug/20 – COVID-19 (SARS-CoV-2) causes prolonged and progressive hypoxia (starving your body of oxygen) by binding to the heme groups in haemoglobin in your red blood cells. People are simply in desaturation (losing o2 in their blood), and that’s what eventually leads to organ failures that kill them, and not any form of ARDS or pneumonia. All the damage to the lungs you see in CT scans are from the release of oxidative iron from the hemes, this overwhelms the natural defences against pulmonary oxidative stress and causes the always bilateral ‘ground glass opacity’ appearance in the lungs, same appearance as acute pneumonia, yet acute pneumonia typically affects only one lung.
Patients returning for re-hospitalization days or weeks after recovery suffering from apparent delayed post-hypoxic leukoencephalopathy (DPHL), which strengthens the notion COVID-19 patients are suffering from hypoxia despite no signs of respiratory ‘tire out’ or fatigue. The use of ventilators is doing far more harm than good.


COVID-19 symptoms are far more in line with flowing ailments than any known viral pneumonia.
How COVID-19 works on the body; its glycoproteins bond to the haeme and in doing so the special and toxic oxidative iron ion found inside the haeme is “disassociated” (released). It’s basically let out of the cage and is now freely roaming around on its own.

This is bad for two reasons;
1. Without the iron ion, haemoglobin can no longer bind to oxygen. Once all the haemoglobin is impaired, the red blood cell has essentially no ability to store its ‘cargo’. It is useless and just running around with COVID-19 virus attached to its porphyrin. All these useless haemoglobin running around not delivering oxygen is what starts to lead to oxygen desaturation, and eventually hypoxia. The affected haemoglobin is permanently stripped of its ability to carry o2 because it has lost its iron ion. The body compensates for this lack of o2 carrying capacity and deliveries by having your kidneys release hormones like erythropoietin, which tell your bone marrow factories to ramp up production on new red blood cells with freshly made and fully functioning haemoglobin. This is the reason you find elevated haemoglobin and decreased blood oxygen saturation as one of the 3 primary indicators of whether a particular patient will soon deteriorate.

It is INCORRECT to assume this traditional Acute Respiratory Distress Syndrome (ARDS) and in doing so, you’re treating the WRONG DISEASE.

2. Millions of iron ions released from their haemes, are now floating through the blood freely. This type of iron ion is highly reactive and causes oxidative damage. While this happens to a limited extent naturally in our bodies, we have clean-up & defence mechanisms to keep the balance. The lungs, in particular, have 3 primary defences to maintain “iron homeostasis”, 2 of which are in the alveoli, little sacs in your lungs.
 The first of the two are little macrophages that roam around and scavenge up any free radicals like this oxidative iron. The third is a lining on the walls (called the epithelial surface) which has a thin layer of fluid packed with high levels of antioxidant molecules, including things like ascorbic acid (Vitamin C) among others. This is usually good enough for naturally occurring rogue iron ions, but with COVID-19 running rampant in your body it’s just too much iron and it begins to overwhelm your lungs’ countermeasures, and thus begins the process of pulmonary oxidative stress.
This leads to damage and inflammation, which leads to all that nasty stuff and damage you see in CT scans of COVID-19 patient lungs.
I believe we are approaching this disease from the wrong angle, and it’s not a viral pneumonia, but rather Hypoxia due to low oxygen levels, or Leukoencephalopathy with Vanishing White Matter (VWM).

-Leukoencephalopathy with vanishing white matter;
-Adulthood Leukodystrophies

Symptoms and signs of COVID-19 correlating to Leukoencephalopathy with Vanishing White Matter (VWM);

-Iron ions overwhelm the lungs and leads to damage and inflammation, which leads to all that nasty stuff and damage you see in CT scans of COVID-19 patient lungs.

-More progressed patients show weakness in deglutition, leading to spastic coughing fits due to inhaled saliva.
-loss of motor functions, irritability, vomiting, coma, and fever has been tied to VWM.

-Loss of smell and taste. Until now, health authorities like the WHO have said a fever, dry cough and fatigue are the symptoms to watch out for. Not entirely correct. As per usual.
-Notice that the symptoms are always bilateral (both lungs at the same time). Pneumonia rarely ever does that, but COVID-19 does every single time.

-Eventually, if the patient’s immune system doesn’t fight off the virus in time before their blood oxygen saturation drops too low, ventilator or no ventilator, organs start shutting down. No fuel, no work.

-Seizures akin to epilepsy in advanced patients have been reported, and muscular spasticity has been observed as well. It would be prudent to not rule out the possibility of SARS-CoV-2 breaching the blood brain barrier and potentially having an effect on neurological and/or the nervous system.  

Why Hydroxychloroquine with Azithromycin Works;

How does chloroquine work? Same way as it does for malaria. Malaria is a parasite that enters the red blood cells and starts eating haemoglobin as its food source. The reason chloroquine works for malaria is the same reason it works for COVID-19 (while not fully understood), it is suspected to bind to DNA and interfere with the ability to effect haemoglobin. The same mechanism that stops malaria from getting its hands on haemoglobin and gobbling it up seems to do the same to COVID-19 from binding to it.

On top of that, Hydroxychloroquine (an advanced descendant of regular old chloroquine) lowers the pH which can interfere with the replication of the virus. Again, while the full details are not known, the entire premise of this potentially ‘game changing’ treatment is to prevent haemoglobin from being interfered with, whether due to malaria or COVID-19.

The legacy media and armchair pseudo-physicians are stating that malaria is bacteria, while COVID-19 is virus, and anti-bacterial drugs don’t work on viruses. They simply don’t understand that a drug doesn’t need to directly act on the pathogen to be effective. Sometimes it’s enough just to stop it from doing what it does to haemoglobin, regardless of the means it uses to do so. The MSM’s claiming that Hydroxychloroquine doesn’t work (purely for political reasons) will be the Fake News blunder to rule them all.

HIV medicine has shown to be effective;

The drug treatment includes a mixture of anti-HIV drugs lopinavir and ritonavir, in combination with flu drug oseltamivir in large doses. Thai doctors have seen success in treating severe cases of the new coronavirus with combination of medications for flu and HIV, with initial results showing vast improvement 48 hours after applying the treatment, including one 70-year-old Chinese woman from Wuhan who tested positive for the coronavirus for 10 days.  While it’s not a cure, it seems to hinder COVID-19 effect on hemoglobin, and gives the immune system a chance to get back in the fight.

A firsthand account (video) from an NYC ICU Doctor describing actual symptoms he is observing – 31/03/20;

“None of it makes sense in the context of a viral pneumonia”…“Symptoms resemble altitude sickness”



Treating patients with the iron ions stripped from their haemoglobin (rendering it abnormally non-functional) with ventilator intubation is futile; unless you’re just hoping the patient’s immune system will work its magic in time. The root of the illness needs to be addressed. The patient’s lungs aren’t ‘tiring out’, they’re pumping just fine. They are starved of oxygen and begin the process of pulmonary oxidative stress.

-There is neither ‘pneumonia’ nor ARDS. At least not the ARDS with established treatment protocols and procedures we’re familiar with. Ventilators are not only the wrong solution, but high pressure intubation can actually wind up causing more damage than without, not to mention complications from tracheal scarring and ulcers given the duration of intubation often required.

-They may still have a use in the immediate future for patients too far to bring back with this newfound knowledge, but moving forward a new treatment protocol needs to be established so we stop treating patients for the wrong disease.

-If you HAVE to inevitably ventilate, do it at low pressure but max O2. Don’t tear up their lungs with max PEEP, you’re doing more harm to the patient.

The Genetic Sequencing of COVID-19;

-Multiple bio-labs have run tests on COVID-19 in order to obtain its genetic sequence. They have all returned the same results; that COVID-19 is an entirely new pathogen, and not a mutation.

-The sequence of COVID-19 is approx. 30,000 nucleotides long, It is a spike protein with no less than 4 clearly defined inserts (parts of the sequence of other pathogens ‘copy and pasted’ in to the COVID-19 sequence. Fig. 2) of approx. 2,000 nucleotides long each. This is clearly visible in gene sequencing reports.

-Specifically, COVID-19 appears to contain inserts of the gene sequence of bat SARS, Ordinary SARS, and HIV, (the GP120) protein. This allows the virus to lock in to the body’s Angiotensin Converting Enzyme 2 (ACE2) receptors very well, making it 1,000x more virulent than ordinary SARS. The HIV element allows it to fly under the immune system’s radar, in the same manner AIDS does.

-Furin moves to the surface of the cell, also contributing to making the virus far more virulent than SARS. -The virus has also been designed to attack multiple different tissue receptors at the same time.

Gene modelling/sequencing of SARS-CoV-2;

Modelled homo-trimer spike glycoprotein of SARS-CoV-2 virus. The inserts from HIV envelop protein are shown with coloured beads, present at the binding site of the protein.
Multiple sequence alignment between spike proteins of 2019 nCoV and SARS. Note; no less than 4 clearly defined inserts.